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Tsai-Turton & Lauderer in their publication in the journal Endocrinology reported that evidence proves oxidative stress (inflammation) induces apoptosis (cell death) in preovulatory follicles and that the antiapoptotic (preventing cell death) effect of FSH is mediated in part by stimulation of follicular glutathione (GSH) and suppression of ROS production

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The extent to which a once-weekly, continuously present amylin analogue reproduces these meal-linked effects as opposed to producing a tonic, non-pulsatile signal quite unlike physiological amylin is a genuinely open question, discussed further in our review of how cagrilintide affects gastric emptying and postprandial metabolism

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