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Proposed explanations for this difference include variations in residual -cell function, glucagon regulation, and baseline body composition

Proposed model for LB nucleation and growth The existing LB models, namely (1) the aggregation/fibrillization theory and (2) the liquidliquid phase separation model, largely theorize monomeric Syn solely as a precursor with fibrillization as the pathological endpoint, ultimately, LB formation

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doi:10.1097/00004703-200604002-00015