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Our findings provided a resource atlas for understanding the diverse pathways PT cells utilize in response to DSA+ status, contributing to ABMR after renal transplantation
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coli proB , and the resulting strains were W303-1b/gsh1 ( GSH1 deletion), W303-1b/gsh1/pro1 ( GSH1::P gap - PRO1 - T pgk1 ) and W303-1b/gsh1/proB ( GSH1::P gap - proB - T pgk1 ) (Table 2)
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