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10.1016/j.abb.2005.11.010 102 KrentzA
Glucagon-like peptide-1 receptor knockout mice are protected from high-fat diet-induced insulin resistance
176 Collectively, these advances underscore a fundamental conceptual shift: proteostasis must now be viewed not as a collection of isolated degradation mechanisms but as a coherent, adaptive, and deeply integrated cellular defense network whose resilience determines disease trajectory
Regulates blood sugar by triggering insulin release after meals
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