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GLP-1R agonism has well-established actions to reduce glucagon secretion in both isolated human islets 28 and in vivo 29 , whereas GIPR agonism increases glucagon secretion in preclinical models 11 and in humans 30
Sin embargo, estas conversiones dependen de la microflora y de la afinidad de las diferentes aminas cuaternarias por las poblaciones microbianas intestinales
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