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Ferroptosis is a form of cell death independent of apoptosis, which relies on iron ions and ROS to induce lipid peroxidation (70)
In the literature on tendon repair, BPC-157 is described as promoting processes that include effects on cell survival and migration, suggesting that it may support cell behaviors necessary for organized tissue repair[1]
There are conditions however that, at the very least, would indicate significant quantitative differences in the sensitivity of humans to these effects, with rodents being considerably more sensitive to thyroid effects by virtue of differences in the biology, transport and control of thyroid hormones in these species as opposed to humans where turnover is appreciably lower and where serum transport of T4/T3 is different to that operating in rodents
BPC-157 supports blood flow and local repair via VEGFR2
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