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Substrate promiscuity and hyperoxidation susceptibility as potential driving forces for the co-evolution of Prx5-Type and Prx6-Type 1-Cys peroxiredoxin mechanisms
Similar to the systemic calcium signal transmission upon GSH treatment, Glu-induced [Ca 2+ ] cyt propagation in systemic leaves were strongly inhibited in GCaMP3/glr3.3 while remained unaffected in GCaMP3/glr3.6 (Supplementary Fig
NAC plays a crucial role in this process by providing a source of cysteine, which is a limiting factor in glutathione production
And this is something, and I know you've explained this to me before, I just can't wrap my head around this, actually with anything really
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