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In this study, IL-1 was used to induce ER stress and subsequent apoptosis
Semaglutide works by mimicking the incretin hormone GLP-1, binding to receptors in the pancreas to stimulate insulin release and in the brain to reduce appetite
Pavlinov et al., 2020)
A consistent pattern emerges in phenotypes with a high metabolic burden, such as obesity and IGT or pre-diabetes, improvements in fasting glucose, OGTT curves, and HbA1c are more pronounced, with some subjects even reverting from IGT to normoglycemia
These are the situations where semaglutide is not safe
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