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This review is focused on the crucial antioxidant role that GSH played in management of OS and its related metabolic disease like T2DM

IGF-1 LR3 contains a 13-amino-acid N-terminal extension and a glutamic acid substitution at position 3, reducing binding affinity for IGF-binding proteins (IGFBPs) by approximately 100-fold compared to endogenous IGF-1

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