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But if the good versus evil axis gets out of whack and there are more free radicals in the body than antioxidants, it results in a condition called oxidative stress
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Although previous ex vivo studies employing NCI-H716 cells (a human L cell line) [35] or human ileal and colonic tissues [14] were indicative of a direct effect of metformin in stimulating GLP-1 release, clinical evidence suggests that the augmentation of postprandial GLP-1 secretion by metformin is largely indirect
MET/HGF pathway activation as a paradigm of resistance to targeted therapies
Developmental regulation of apolipoprotein B mRNA editing is an autonomous function of small intestine involving homeobox gene Cdx1
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