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Therefore, exogenous GSH provided a significant suppression of oxidative stress which indicates the antioxidant effects of this compound

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however, aggregates of TTR, including oligomeric forms, have previously been linked to cytotoxicity [81,82,83,84,85] supporting the possibility that misfolded TTR may contribute to this response

In summary, our study establishes that VRK2 expression dictates PC cell vulnerability to GSH-targeted therapies through distinct mechanisms

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