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ARID1A expression is down-regulated by oxidative stress in endometriosis and endometriosis-associated ovarian cancer
Generally though, interest in complement as a potential mechanism to enhance the antiviral activity of nAbs has been limited, in part because of early in vivo observations that nAbs against several viruses had similar levels of protection in complement-deficient mice and wild-type mice 87
Clinical and experimental obstetrics & gynecology
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