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Qian L, Wei W
Resistance training preserves and builds lean tissue during GLP-1 treatment by creating mechanical tension and protein synthesis stimulus that counteracts the medication's appetite suppression and modest lean mass reduction
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This steep Ca 2+ gradient creates a strong driving force for Ca 2+ entry and is maintained by the low Ca 2+ permeability of the resting erythrocyte and high Ca 2+ -extruding capacity through a Ca 2+ ATPase and Na + /Ca 2+ exchanger in the cell membrane
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