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497 Astrocytes, however, also express GDI and vesicle traffic in astrocytes is similarly impaired in the absence of GDI1

Endocrine Reviews, 18(5), 621645

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Critically, integrating genomic and molecular data has uncovered fundamental mechanisms of patient heterogeneity, such as the discovery that a diurnal BMAL1-HIF2A complex regulates myocardial vulnerability to injury, providing a molecular rationale for the long-observed circadian patterns of MI severity and opening the door to chronopharmacologytiming interventions for maximum efficacy (Ruan et al., 2025)