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Thus, we measured TrB1-3 distribution in B cell-specific Gclc -deficient mice and found that TrB1 were increased in Gclc -deficient mice compared to controls (Supplementary Fig
In parallel, proinflammatory cytokines become upregulated in chondrocytes, osteoblasts, and synovial fibroblasts, activating mitogen-activated protein kinase (MAPK) and nuclear factor B (NF-B) signaling
Electrophysiologic analysis of tight junction size and charge selectivity
Based analytical procedures to evaluate diabetic biomarkers and related parameters
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