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Here we show that HGF adopts an enhanced form with dynamically increased receptor-binding affinity and nitration-dysfunction resistance through interaction with lipoic acid trisulfide (LASSS) under physiological conditions

( GAP1 expression from its native promoter would not serve as a good control here because pGAP1 is repressed by the ammonium sulphate N-source and so D-HIS would not be taken up by the cells)

Nat Commun 15(1):3113

These observations were performed by three independent physiatrists who were blinded to the treatment groups, ensuring unbiased assessment