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Plated cells were pretreated with 4 L of agonist, or antagonist (Ex 9-39 (GLP-1R antagonist) or BIIE0246 (Y2-R antagonist)), at a given target concentration and incubated for 20 min before performing the assay
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Preclinical evidence has suggested that GLP-1R agonists can suppress hepatocellular inflammation and increased oxidative stress indirectly through several cellular pathways that are, in part, independent of weight reduction, since hepatocytes, Kupffer cells, and stellate cells do not express GLP-1Rs [7985]
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