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The experiment included detailed clinical, morphological, and histological assessments to determine the degree of joint degeneration and recovery following different intra-articular treatments [1]
2024) offered that epithelial-mesenchymal transitions (EMTs) are the culprit, suggesting that acquired resistance is the product of plasticity rather than the outgrowth of pre-existing mutant subclones
That stability profile is the main reason it appears so frequently in preclinical literature
doi:10.17116/terarkh20158712117-121
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