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Philp D, et al
p53 mutation may promote hypoxia-induced genomic instability, leading to the activation of pro-oncogenic signaling such as hypoxia-inducible factor (HIF) 1 [4]
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Moreover, AMPK exhibited a comparable half-life in shCtrl and shGDH1 cells in a cycloheximide (CHX) chase assay (Extended Data Fig
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