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Notably, all available antipsychotic drugs are thought to work mainly through blockade of the type 2 dopaminergic receptor, and this main target has not changed since the discovery of this mechanism 60 years ago [21]
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For example, the variability in ROS levels among different types of CSCs raises questions about whether diminished ROS levels can entirely explain the preservation of the stem cell phenotype, particularly in light of the altered glucose metabolism characteristic of these cells [53, 54]
M.et al (1996)
