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The restoration of mTOR signaling with Fer1 rescue treatment in the context of RSL3 effects, and the genetic knockdown of GPX4, reaffirm our in vitro findings that RSL3-mediated induction of ferroptosis, mTOR pathway suppression, DNA damage and failure of DNA repair, and autophagy are GPX4-dependent processes in thyroid cancer cells
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Its primary role is neutralising free radicals - unstable molecules that damage cells, accelerate visible ageing, and contribute to various diseases
Redox regulation of hormone sensitive lipase: potential role in the mechanism of mehp-induced stimulation of basal steroid synthesis in MA-10 Leydig cells
