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GLP-1based drugs delay gastric emptying and may affect oral absorption of other medications
A review of changes in anthropometric indices between studies revealed conflicting results in animal studies [42,43,44]
Activation of these receptors influences insulin secretion, slows gastric emptying, and contributes to satiety signals following nutrient intake
Contrary to expectations, glycaemic control was also improved after chronic treatment with the GLP-1R/GcgR co-agonist, which implies that GLP-1 activity protected against glucagon-induced hyperglycaemia [17]
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