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10.1074/jbc.M508361200 J

It may take time, but I hope we do. Gwen Allen, Dongjune Lee and Seungeun Hong contributed to this article

In many in vivo disease studies or oxidative stress stimulation models, GPX1 deficiency enhances cell damage, apoptosis, and even leads to cell death

These include its comparatively high utilization of oxygen and therefore generation of free radical byproducts, its modest antioxidant defenses, its lipid-rich constitution that provides substrates ready for oxidation, the reducing potential of certain neurotransmitters, and the presence of redox-catalytic metals, like iron and copper (47)

Lung disease (chronic obstructive pulmonary disease)