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Taken together, these findings suggest that metformin may protect chondrocytes from H 2 O 2 -induced damage, potentially through activating Nrf2 signaling pathway

In contrast, mutation of the C-terminal active site cysteine increases the activity of GLRX, presumably by preventing disulfide bond formation between the two active site cysteines 20,21 , and renders GLRX a monothiol enzyme only capable of catalysis of (de)glutathionylation 22

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