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Modulation of enzymes involved in NAD + consumption and synthesis In addition to NAD + precursor-based strategies, halting of processes linked to NAD + consumption or biosynthesis represents a complementary and therapeutically appealing approach to alter or restore NAD + homeostasis in context-specific applications
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Identification of mutations in the parkin gene as a genetic cause of autosomal recessive juvenile parkinsonism (ARJP)
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