Amplifying pro-survival signaling in a genomically primed context The functional contribution of ER to EAOC pathogenesis is best understood not as a primary oncogenic driver, but as a multifaceted modulator that potentiates pro-tumorigenic phenotypes
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References 09738086 Treiman DM, Meyers PD, Walton NY, Collins JF, Colling C, Rowan AJ, Handforth A, Faught E, Calabrese VP, Uthman BM, Ramsay RE, Mamdani MB
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