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FIGURE 1 3 Excitotoxicity in TBI The disruption of the BBB resulting from TBI leads to the release of excessive neurotransmitters and a breakdown in the normal glutamate reuptake by transporters (Chamoun, Suki, Gopinath, Goodman and Robertson, 2010)

Mechanism (ii) from Fig

The D-Retro-Inverso modification uses D-amino acids in reverse sequence, ensuring resistance to proteolytic degradation while preserving the capacity to interfere with the target protein interactions

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Due to its critical function in renewing nerve fibers and mending nerve damage, vitamin B12 is essential for maintaining healthy nerves