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Research into MT-II focuses on its potential to treat skin disorders, including vitiligo, and to explore its role in UV exposure protection

125 This is exemplified by immune evasion via antigen loss variants under immunotherapy or secondary mutations in driver oncogenes (e.g., EGFR-T790M, ALK-L1196M, BCR-ABL-T315I) in the context of targeted therapies

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6 Integrative discussion and future perspectives The intricate interplay between mitochondrial dysfunction and pathogen infection highlights mitochondria as a central hub for host-pathogen interactions