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Most importantly, this protective phenotype appears to involve the following beneficial cellular adaptions: (i) enhanced metabolism of ethanol and acetaldehyde, (ii) suppression of lipogenic genes and induction of genes involved in fatty acid oxidation, and (iii) induction of the NRF2 antioxidant response and the AMPK metabolic signaling pathway
It is not indicated for type 1 diabetes or diabetic ketoacidosis
Accordingly, peri-pancreatic fat served as the most reliable site to evaluate inflammation, where vehicle-treated mice displayed abundant CD45 cells that were substantially reduced by all incretin-based treatments
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