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Abscisic acid and glycine betaine mediated tolerance mechanisms under drought stress and recovery in Axonopus compressus : a new insight
The primary therapies currently involve managing blood glucose levels, blood pressure, and blood lipid levels, diet, exercise therapy, and corresponding treatments depending on the site of microvascular complications
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However, the mechanism beneath the effect of GSH depletion-induced oxidative stress in RPE cells and the subsequent RPE damage is not well established
Mechanistically, ginger's active compound, [6]-shogaol, has been identified as a mediator of vasodilation, primarily through its action on calcitonin gene-related peptide (CGRP) pathways and subsequent activation of endothelial nitric oxide synthase
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