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doi:10.1016/S0025-6196(11)63007-3
10.1186/s12943-020-01258-7 Summary Keywords ulcerative colitis, GHK-Cu, SIRT1/STAT3 pathway, co-culture system, network pharmacology, molecular docking Citation Mao S, Huang J, Li J, Sun F, Zhang Q, Cheng Q, Zeng W, Lei D, Wang S and Yao J (2025) Exploring the beneficial effects of GHK-Cu on an experimental model of colitis and the underlying mechanisms
The conversion of T4 into the more physiologically active T3 form is lowered due to a reduction in the activity of these deiodinases, which occurs when Se is deficient [33]
Therapeutic potential, pharmacological activities and analytical aspects of liriodendrin in medicine: novel and potential therapy options for the treatment of human disorders and associated complications
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