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is supported by a Department of Medicine, University of Toronto Merit Award and receives support from the CIHR, Diabetes Canada, and the Heart and Stroke Richard Lewar Centre of Excellence and the Heart and Stroke Foundation of Canada
It mainly consists of three parallel mechanisms, i.e., extracellular signal regulated kinases (ERKs), c-Jun N-terminal kinases (JNKs) and p38 MAPK 43
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95% CI, 0.99 to 1.36) between two users, but SGLT2i users had a significantly lower risk of proliferative DR (SHR, 0.53
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