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This is likely due to a reduction of peptide interactions (mainly A5 Cagri ) with ECL3 that is a consequence of the engagement of the lipidated N-terminus with the top segment of TM5/ECL2, facilitating flexibility of ECL3
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A study published in The Lancet evaluated GLP-3 in 281 adults with type 2 diabetes who were receiving standard treatment with metformin
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