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Caught in the cROSsfire: GSH controls T cell metabolic reprogramming

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Five impact areas are identified: glycemic profile, inflammation, oxidative stress, lipid profile, and hormonal profile, each linked to metabolic and endocrine alterations characteristic of PCOS

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281 In cancer cells, sustained or rewired UPR activation promotes survival under chemotherapy and targeted therapy by enhancing chaperone expression, attenuating global protein translation, limiting oxidative stress, and suppressing apoptosis through the ATF4CHOP axis