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We have demonstrated that during viral infections a decrease of intracellular GSH is operative that varies in intensity, duration and mechanism of induction depending on the type of virus and the infected host cell ( Figure 3

Animal models for dry eye such as desiccating stress and lacrimal gland excision and cataract including selenite- or steroid-induced forms are well-established

ATP increases within the lumen of the endoplasmic reticulum upon intracellular Ca2+ release

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