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Receptor mapping of these contact pairs revealed that transducer binding resulted in a network of contacts at the lower half of GLP-1R shared across agonist-bound structures

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Macrophages facilitate this activation by releasing pro-fibrogenic mediators, including TGF-1, VEGF, and galectin-3 (60)

A short follow-up may not fully capture the long-term recurrence patterns of the disease

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