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Hyperinvasive meningococci induce intra-nuclear cleavage of the NF-B protein p65/relA by meningococcal igA protease
Interest in GIPR agonism for the treatment of obesity and diabetes was recently propelled by the clinical success of unimolecular dual-agonists targeting the receptors for GIP and GLP-1, with reported significantly improved body weight and glucose control in patients with obesity and type II diabetes
It converts it back to its active, reduced form (GSH)
Iron drives T helper cell pathogenicity by promoting RNA-binding protein PCBP1-mediated proinflammatory cytokine production
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