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2 Secondary First Start with desk research to surface what's already known
[1] [5] These medications bind to GLP-1 receptors primarily in the pancreas, brain, and gastrointestinal tract, leading to increased insulin secretion, reduced glucagon release, and enhanced satiety
doi:10.14309/ajg.0000000000003525
GLP-1 agonists enhance insulin secretion and suppress glucagon release only when blood glucose levels are elevated, a glucose-dependent mechanism that substantially reduces the risk of hypoglycaemia compared to other diabetes medications such as sulphonylureas
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