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Increased TMAO levels in addition to endothelial dysfunction, vascular aging, and oxidative stresses have been shown to cause thrombotic events and platelet excitability in humans, which in turn induce atherosclerosis [69, 72]
Pathway 2: Nitric Oxide System Modulation Independently, the BPC-157 peptide interacts with the nitric oxide (NO) signaling system a key regulator of vascular tone, inflammation resolution, and cellular repair signaling
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