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On the other hand, exenatide did not affect endothelial function in rat conduit arteries when tested ex vivo after intralipid infusion [107, 108]
4C), confirming preserved motor function throughout the experiment
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Gut microbiota dysbiosis contributes to HTN through mechanisms involving OS and inflammation (Li et al., 2021)
These agents target complementary pathways in atherosclerosis, including lipid lowering, metabolic regulation, and reduction of cardiovascular risk
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