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Methods: PBC patients followed in a tertiary hepatology Italian centre with available quantitative AMA-M2 values were included in a cross-sectional analysis
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Hydrogen sulfide biochemistry and interplay with other gaseous mediators in mammalian physiology
Oxidative-stress related toxicity in hepatocytes can be prevented by enhancing LDs formation
According to our current knowledge the most likely scenario is that AITC treatment causes ROS production 19,20 and induces the expression of several GSTs 17,40,41 as part of the plants detoxification system to conjugate AITC to glutathione leading to its intracellular depletion
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