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Because of the hypothesised abnormalities in oxidative stress and suggested links between initial inflammation, glutamate excitotoxicity and neuronal dysfunction/loss [25], our additional interest was in, GSH, glutamate and glutamine, and total choline as biomarkers of these processes

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Previous studies that have measured the incorporation of cysteine into metabolites suggested that additional sources of cysteine for tumours, beyond extracellular cystine or de novo synthesized cysteine, probably exist 16

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