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Ziegler, D., Nowak, H., Kempler, P., Vargha, P., & Low, P

Additionally, diosgenin sensitized colorectal cancer cells to apoptosis induced by TRAIL via activation of the p38MAPK pathway, overexpression of death receptor-5 (DR5), and downregulation of the Akt pathway (245)

The change extends the proteins half-life and permits it to disrupt normal FOXO4 function

Caspase-1 cleaves the N-terminal structural domain of GSDMD, and the cleaved N-terminal fragment inserts into the cell membrane, to form pores, ultimately causing membrane rupture and the release of pro-inflammatory intracellular contents

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