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According to the evidence discussed in this paper, we can hypothesize that chronic PE can modulate the etiological factors related to the onset of this condition which involves a high and chronic ROS production, a deficient EAS enzymatic activity, and a pro-inflammatory autoimmune response (Xie et al., 2016)
However, based on current research data, metabolic factors cannot be ruled out as potential contributing factors
We had a relatively large sample size
A recent systematic review indicated that while reductions in fat mass and visceral adipose tissue are generally consistent in obese or overweight adults, changes in lean mass are variable, showing decreases, preservation, or increases [49]
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