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[4] [5] Semaglutide works by mimicking the incretin hormone GLP-1, which stimulates insulin secretion in a glucose-dependent manner, suppresses glucagon release, slows gastric emptying, and reduces appetite through central nervous system pathways
GLP-1 receptor agonists were originally approved for the treatment of type 2 diabetes, though given the prevalence of GLP-1 receptors in various peripheral tissues, more recent studies have started to look at additional etiologies for which GLP-1 agonists could be a feasible therapeutic option, including MASLD
The study measured weight loss at 24 weeks and 48 weeks
These drugs have the potential to decrease symptom burden, enhance mobility, and increase quality of life while integrating with existing treatments such as compression and manual therapies
General methodological considerations for the assessment of nutritional influences on human cognitive functions
