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The data on rhIGF-1 support the mechanistic rationale that an IGF-1 analog like LR3 (which has extended half-life and greater receptor availability in research settings) could enhance protein synthesis, stimulate cellular proliferation pathways and promote more efficient nutrient partitioning, though direct clinical trials of IGF-1 LR3 itself in humans are lacking
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Its unknown whether the drug raises the risk of these types of tumors in people, too
Your body has never experienced pharmaceutical glucagon receptor agonism at these potencies
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