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Further studies are needed to understand the biological mechanisms, if any, between GLP-1 RAs and breast cancer outcomes
The mechanism appears to involve modulation of stress response pathways that are hyperactive during withdrawal
Below is a balanced view that reflects both research findings and clinical experience
Emerging research shows that certain gut bacteria can promote the release of GLP-1 through several important mechanisms, including the production of beneficial metabolites, like short-chain fatty acids(SCFAs) that stimulate GLP-1 secretion from colonic cells