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Thus, through genetic manipulation of GSH biosynthesis, the present study, along with the BSO study, implicates a role for chronic GSH depletion in protection against ethanol-induced hepatotoxicity
Oral glutathione is subject to degradation by digestive enzymes, and early studies found that a single oral dose of up to 3,000 mg failed to increase plasma glutathione levels in healthy volunteers [1][13]
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INTRODUCTION Ethics statement Ethics approval was not required for this in vitro study, as it did not involve human participants, identifiable human data, or animal subjects
