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A genetic study found that the interstitial deletion of chromosome 4q in a child with autism contributed to hemizygosity of the glutamate receptor AMPA2, glycine receptors GLRB and GLRA3, and neuropeptide receptors NPY1R and NPY5R [75]

FIGURE 3 3.3 GHK-Cu promoted mucosal healing GHK-Cu treatment significantly enhanced of ZO-1 and Occludin expression in colitic mice, which were downregulated by DSS (Figures 4AC)

The localization of the cystine/glutamate transporter could not be confirmed by commercially available antibodies because of their low sensitivity and specificity

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