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ABSTRACT: ClinicalTrials.gov identifier: NCT04105504
Fundamental Mechanisms of Regulated Cell Death and Implications for Heart Disease
Free Cu2+ is toxic (Fenton reactions, hydroxyl radicals), but the GHK-Cu complex is stable, diffusible and delivers copper in a targeted manner to copper-dependent metalloenzymes: lysyl oxidase (collagen/elastin crosslinking), Cu/Zn superoxide dismutase (SOD1, antioxidant defence), tyrosinase (controlled melanogenesis), dopamine-beta-hydroxylase
2h, Extended Data Fig
Pills could expand the market into earlier treatment stages, primary care, emerging markets, and long-term maintenance
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